Origin of Keratoconus sickness

After years of researches about the causes of the keratoconus I and my team have found the ratio for a possible infective origin of the sickness.
We have to classify the keratoconus as the conclusive anatomical dystrophy of the corneal collagen layers caused by an infective sub acute chronic keratocongiuntivitis more or less frequently found into one or more members of a family.
Symptoms like burning, sensation of foreign body, need of “rubbing” and in many cases an eye dryness, due sometimes to the use of medicine and/or the use of substances for the care and sterilization of contact lenses, both soft and semi-rigid, clearly show a sickness of corneal collagen but with common characteristic of many other infectious sicknesses of the cornea and of the conjunctive.

The Pathogenous Agent, which we found out in January 2004, represents the beginning moment of the corneal collagen modification that has the capability to alter the strong structural tie of the molecules of the helicoid of the same collagen, in this way allowing the collagen to stretch, simply under the constant action of the intraocular pressure that pushes the cornea growth outside. International literature on the contrary speaks of genetic causes. In fact three different Universities Research Groups (2 in Italy, 1 in U.S.A.) have found 4 different genes that they think responsible of the sickness. To clarify this genetic theory we should understand that in the keratoconus we are speaking of a collagen sickness and because of collagen molecule is not only in the cornea of the eye but it is spread all over the human body, we should have a genetically induced collagenopathy. But in fact they did not find any other organ of the body affected by any collagenopathy as normally occurs for other collagenopaties as “lupus eritematosus systemic”, “arthritis reumatoidis systemic” and so on. Nor the genetic can explain the presence of starting keratoconus in patients aging 50 and over. Or why could be affected only one member of a whole family. Nor they can explain why the sickness start in one eye and the fellow eye will follow years after. Nor the genetic theory can explain why and how we have 45% of a relapsing keratoconus in the transplanted layers of total or lamellar corneal transplants.
On the contrary all these facts can be easily explained by an infective pathogen. In fact from January 2004, with my team, we are convinced to have discovered the Pathogenous Agent that we found to be a parasite which has been electronically diagnosed and it is constantly present in 99,9% of the keratoconus examined patients and we believe it responsible for the whole pathology.
We also discovered the co-factors responsible of the arrival of the pathogenous agent in the patients’ eyes causing the infection and then the sickness which is in progressive expansion into the population.

This “initial biological infectious” stage is responsible of those bio-mechanical de-structuring modifications of the normal collagen corneal tissue: only in this way we can explain the passage from the pre-clinic asymptomatic stage of keratoconus to stage I, then II, then III until to the spontaneous corneal perforation in the most severe cases.
Therefore it is needed an anatomical geometrical Asymmetric restructuration of the cornea and this is from more than 20 years possible through my incisional technique ARK-Mini ARK that, giving an implosive restructuration (in our 20 years follow-up we have constantly seen an increase in central and paracentral thickness from 20 to 80 microns) to the affected ectasic cornea, can prevent any further development till the need of the corneal transplant. The same technique is the only one capable to correct at the same time the 80% of refractive related disorders allowing the 98% of the patients to see the 80% without glasses and the remaining 18% with a useful use of simple glasses to see in a way according to the needs of their life.
Last, we are already making preventive medicine examining the members of the family of the patients affected by keratoconus finding out the not yet diagnosed keratoconus patients and stabilizing, with our Bioelectronic therapy, those patients affected by early stage keratoconus that can still see without correction, avoiding any possible surgery in their future.